Episode 535: Dr. Stephanie Venn-Watson: The Longevity Nutrient That Could Slow Aging and Protect Your Cells

Pressure it. - All right, you guys, welcome to another episode of "Happets and Hustle." And we have a real change maker, even CNBC called her that. This woman, her accolades are just extraordinary and super impressive, and her name is Stephanie Van Watson, and she's joining us today.

So oh, she has a new book out coming out and actually her new book is called The Long Jevity Nutrient, and it's coming out on paperback, you said. - That's right, and March, so very excited. - Amazing. - Well, congratulations, and thank you for being here.

- Thank you, Jennifer. Fantastic. - Thank you very excited. - I buy the way. I feel like we just did a whole podcast before the podcast, which is why I try.

“That's why I felt like I liked you, and I'm like, oh, shoot, we're in trouble, because I knew”

that it would take about an hour to get into the room, and it kind of did. And I didn't want to ask you any real pertinent questions, because I wanted to make sure I asked you on the show, but so anyway, thank you for being here, and your background is quite impressive. I mean, it really is.

Do you want to kind of give people the origin story of why you're even here right now? - Sure, sure. So I was out of the womb, just going to see it all started in 1971, or whatever, you know?

- That was, yeah, so I've always been a self-proclaimed nerd.

I've always been loving, finding patterns, pattern recognition is just a natural thing. Since I was little, my dad would come home, we worked at a library, and he would bring home books, like, of just a numbers, and I would take him down to the basement, and circle patterns, like, what a nerd, right? - Really?

You are. - They're right. The thing that I would do, so I don't think they were worried at the time. So, fast forward to college, and I, you know, was ready to go for a medical school, and

“then I read a book from Laurie Garrett called the time in plague, and I learned what?”

There's a profession called Epidemiology, where there are doctors, and their whole job is to find patterns, like, this is fantastic, right? Medvedinary Epidemiologist coincidentally, and he said, gosh, if you really want to understand diseases, stuff, become a veterinary epidemiologist, because you'll understand how diseases work in a lot of different species, and then you understand the disease.

So, that's triggered that, went to veterinary school, caught in my epidemiology degree in over at Emory, and, uh, next thing, you know, you're in the Navy. - Yes, there you go, signing up, ready to serve.

- You know, I've never met someone that, actually, you were basically a veterinarian that went

to the Navy and worked on, mostly, just dolphins, but that was basically what you studied on. - Correct? - Right, yeah, that's right. - Yeah, brought in to help, well, like, a huge testament to the Navy race, that they have this population of Navy dolphins that have been there, you know, live in San Diego, base, since the 1960s, to their credit, they brought me in to help the dolphins.

So, it wasn't, the fact that this has turned to it, you know, spinning out to be able to improve human health is great, but this whole thing started because they wanted to bring in a veterinary epidemiologist to study 60 years of health data to continually improve the health of their dolphins. - Well, we don't know, it's going to ask you what I find interesting, is a lot of people who are doing what you're doing in the longevity space who are, you know,

or in general, a lot of the research is based on studying on rodents, or worms, or things

of that nature. I've never heard of people doing research, you know, by accident, let's say,

on a mammal like a dolphin, or a dolphin, and I've never heard anyone working with dolphins to be honest, what is, well, how much more accurate is the findings when you work with something like it, with someone like, well, an animal like a dolphin versus even working with a rodent, because a rodent is a very different species, obviously, than humans, and then people are using that information to translate for humans. - Yeah, it's a great question, Jen. So, you know, the, a lot of the

research is being done on worms and mice and flies, because it's easier, right? - Yeah. - That's the amount of three years lifespan max for the mice, and so you can rapidly do studies for worms it's a week. So it's like, gosh, if I can get a worm to live to two weeks, where I can say we doubled its lifespan, you know, and a huge one on that friend. So it's easy to do that research, but to your point, while there are similarities among all animals, there are really

“important differences, and what the gift that the dolphins gave us was that if you step back and”

look at all this focus in short-lived species, evolution has already figured out how a human can live 37 times longer than a mouse. - Right. - So, right, so it's like, evolution has already figured out how a mammal who has, you know, we both have livers and brains and splings and rib blood cells,

in front of us by putting in the dolphin angle, which is we were seeing that older dolphins were

“aging a lot like older people naturally, not forcing a chronic disease in a mouse and a lab,”

but just seeing all of a sudden we were gifted with this patient population that have large brains and long lives, and we have co-evolved to have similar mechanisms that allow us to live a long time, but also similar mechanisms that help us be susceptible to diseases like high cholesterol and chronic inflammation and Alzheimer's. So it was just a really, a fortuitous gift. - It really is, I mean, so what were your, when you were brought into even work with the dolphin, were you there for a

very specific thing that you were looking for, and then you kind of happenstance found all this other longevity or this main longevity, I guess, piece of information that nobody seemed to have known about, which is kind of crazy to me. - It is. - What was the whole process? Like, what did you kind of learn? What was the whole thing there? - Right, how many years were you there? - Her gosh, she's 20 years and was 20 years. - So you were researching dolphins for the basics, for the,

“to kind of learn about health and longevity in humans, and that basically, or how humans would”

live longer, how do they actually live longer? Like, what was the, what was the basic reason why they brought you in? - Right, so they brought me in because they had accumulated half a century, more than half a century of dolphin health data. So they had, they were so ahead of the times that they were co-routinely collecting blood samples that are part of their routine health care. They had it in electronic database, like data from 1980s, 1990s, and dolphins throughout their

life. So every quarter, imagine going to your doctor, getting a full CBC income, and then all that data being put into electronic database, your whole life, and storing the samples, the serum samples, so that you could actually go back, that allowed us to do this thing called metabolomics, and actually study thousands of small molecules and match them to which my molecules predicted dolphins that got high cholesterol, got chronic inflammation, and more importantly,

“which my molecules protected against it. So they brought me in because, initially, because it was”

about looking at infectious diseases that were present in wild dolphins and to make sure navy dolphins were protected. There's a virus called more bill of virus, which is like measles. It's in fact, it's like, it is measles in dolphins that was causing mass mortality events in wild dolphins, and so they had brought me in to understand, you know, what were the risks to the dolphin

population ended up being very low. But then while I was there over the first decade, then we

started really transitioning to chronic diseases, which I'll be totally honest. I thought, the chronic diseases are boring. Yeah, that's exciting. Like a bullet. And then I got into like, oh my gosh, like this is so this for where we were studying so much more impact positive impact we could have on the dolphins, and then to be able to spin out and see these exact same patterns and the ability to translate it to humans was huge. So the initial studies were all

to help the dolphins. We found that this specific nutrient to a C-15 present in some fish, but not all the dolphins that were getting the fish with a higher C-15, we're getting higher C-15 levels. We now know that this was enabling them to be protected against these aging associated conditions. And the same epidemiological studies were popping up in humans. It's just it was hidden because, you know, it's saturated fat and we thought, well gosh, that doesn't really fit the pattern that we

know. So it was just kind of pushed to the side. So nobody was at nobody really kind of was able to see that finding or look what they know was looking for it really. That's right. And so just by kind of coincidence or just happened stance, you were figured out that it was this molecule called C-15 that was really enhancing the longevity and the health of the dolphins. That's right. And so

we saw the first studies where you know association, okay, they're associated with the dolphins

that were aging healthier. And then and all of this was funded by the Office of Naval Research. So, you know, we're held at the highest standard of these studies. So we said, well gosh, let's go find more fish that have higher C-15 in them. Let's give them a modified diet that has higher C-15 fish. And let's see what happens. And we did it and they got better, you know, wanting Python way. You know, it got so surreal. They were able to. We were seeing,

like, Enemia was completely alleviated. We saw cholesterol going down inflammation going down. And you know, it was changing fish to be fair. But we saw, as we increase our C-15 levels, through this modified diet, they got better. So then from there, we then moved C-15, pure C-15 into the lab. And then spent the next, you know, 10 years, the past 10 years, understanding from a pharmaceutical, you know, approach. How does the molecule work?

And so all of those studies have been undergoing it most importantly. We've been doing the work.

But now, you know, there are groups around the world who are publishing and out over 100 peer-reviewed papers. And average of one new paper coming out on C-15 a week.

“It's such an exciting time. Are you kidding? No. So how do you explain what it is?”

So it's a central fatty acid. Is that what it is? Saturated fatty acid? Right. So C-15, it's an odd chain saturated fat. And, you know, we've been told our whole lives. All dietary saturated fats are bad. We now know 100% unequivocally different, not all saturated fats are equal. So these odd chain saturated fats. So if as C-15, we think it's 15 carbons. So I have, if it has an odd number of carbons, especially C-15, it protects against, you know,

development of metabolic disease or type 2 diabetes and heart disease. If it's an even chain saturated fat, especially C-16. So we're just talking about adding one carbon on the same molecule increase risk of type 2 diabetes. And the exact, I mean, it's mind blowing what nature's doing with two with just adding one carbon. And we know this, not because I'm saying it, it's because of the preponderance of data that have been published showing over and over again. Meta analyses

of these large prospective cohort studies and populations throughout the world showing this over and over again. Well, I found so interesting is that without enough C-15, your age is accelerated at a very fast pace. And you hear about all these other longevity medications or supplements, like I was saying to you, like metformin, reprimisen, they say that also's another one.

“The other one I couldn't pronounce asked a zanthin. I think that was another one. Yeah, that's”

I said it. Yeah, yeah. He will leave. I said I can remember how to say it, if I could never pronounce

it. What is the difference between all these other supplements, medications and and C-15, right? So what's exciting is science. We have all of a sudden, we have a forest for longevity and anti-aging is going from, you know, fiction to or from, you know, from science fiction to science about. And so we now know their specific longevity regulating pathways. It's called the human, a longevity regulating pathway. And it details those pathways. Like if you tap into that this pathway,

then you this will lead to longer-lived mice and worms and dogs and humans. So we also know that there are these hallmarks of aging. So ways that, you know, define how we age at the cellular level. And if we can target these hallmarks, then again, we could live longer. So when we look at the traditional approach to longevity enhancing candidates, reprimisen's at the top. And it's because it targets this almost the heart of the longevity regulating pathway. So much so that, you know,

it's job is to inhibit M-tor, which then has this, you know, enables longevity, M-tor stands

“for the mechanistic M-t target of raffamisen. So it's so important. It's so important to longevity.”

It has its own pathway named in it. So, so that's raffamisen. We talk about metformin. This does the heart of the longevity pathway. So it activates M-t-k. And by doing that, again, this is a huge longevity enabler. It has the added bonus of also. This is the mechanism that, you know, this is how metformin also helps improve glucose control. So you kind of get a good one to hit. So when you talk about these various longevity enhancers, they do provide a lot of promise because

they're foreign molecules that tap into the longevity pathway. And are showing a lot of promise. C-15 comes in right out of nowhere. And it's like, oh, it's in breast milk and butter.

It's basically found in dairy products, which is, you know, what, why it's kind of

concerning to me is because we've been, well, dairy has been vilified. I feel in the media. That's the first thing. People are drinking now oat milk instead of drinking regular milk. And if you look at the ingredients of oat milk, it is literally like having a Snickers bar. There's more sugar and shit in that in those in the oat milk. I don't care what brand it is. I don't care what it is. It's not a healthy option. But we've been now with, with marketing and branding and just rep,

like repetition, we're brainwashed to believe that oat milk is better than dairy, which is, I mean, even, I even drink almond milk, even though I know even almonds are like stuff with all

what do you call it when they, when they spray the almonds and all the, all the things. My point is

“we're, we're substituting dairy for all these other alternative milks. When there's so many health”

benefits to dairy, right? That's right. Unless your lactose intolerant, right, which a lot of people, you know, a lot of people are, but you, the whole point that you're making, Jen, is that, you know, we, that sea 15, the wild dolphins were getting their sea 15 from fish, our primary source. Our five humans primary source. Yeah. My of our is dairy fat so much so that for a long time, sea 15 has been used as a biomarker of how much dairy fat we eat. Even before we knew there

were any benefits. Wow. So it's like, how much dairy fat we dictate how much sea 15 we have. We have.

So that, um, you know, so now we know that because we've been decreasing our intake of sea 15,

that our sea 15 levels have been decreasing, the same thing had been happening in the dolphins, right, because they've been moving away from a sea 15-rich diet after the 1990s. But we're that eating. So they had actually, before they were eating, you'll look on fish, which was this, is a super fatty fish. The you'll look on the fishery died out. And so they had to move to a less fatty fish. So at the same time, we were moving away from whole that milks. They were

moving away from their whole that fish. And so we had a coinciding 50-year experiment between two long-lived, archboring mammalian populations. And what we started seeing is we started seeing this, you know, rise in aging associated diseases and dolphins, including fatty liver disease,

you know, non-alcoholic, which showed up in humans in 1980 for the, you know, the first cases

in our present and one in three people globally. So we had these parallels, really interesting, parallels happening. And that's where we learned that as our sea 15, one of the core roles of sea 15

“is it helps to, it's a sturdy fatty acid that as essential in strengthening our cell membranes.”

So it literally strengthens our cell membranes against what's called lipid proxidation. What we now know is that if our sea 15 levels get below, like about 0.2% of total fatty acids, it's like arm, you know, bricks inside of it. If we don't have enough bricks, that cell membrane becomes fragile. And it causes what you had talked about. This new form of cell death called pharoptosis, this was discovered by research researchers at Columbia University

back in 2012. There've been over 20,000 papers written on this new form of cell death. We know it accelerates aging, so that's the, you know, accelerating component. We know it accelerates the onset and the exacerbation of type II diabetes, heart disease, cognitive decline. What nobody's been able to figure it out is where did it come from? Why all of a sudden, our cells dying in this whole new way? And that's where the dolphins taught us that sea 15 deficiency

in the diet causes pharoptosis. And more importantly, it's fixable. We can actually reverse it just like sea or vitamin C deficiency and scurvy and vitamin D deficiency in rickets. This is just it's the nutritional deficiency of our generation, you know, of our time. You know, so, so if people are not eating dairy, does that mean that they're, they're most likely deficient in sea 15? Yes. Yes. Yes. So we'll probably like, a big portion of the population now are just

deficient and they, they may have a lot of these health concerns and not even really know the root cause because they're not even looking for it. That's right. And what we're seeing is, you know, we're seeing younger people getting more aging, getting aging associated diseases faster. We work with Dr. Jeff Schwimmer and he's a pediatrician. He works with, um, and with fatty liver disease. And he's been on the forefront of fatty liver disease and kids. And Jeff, you know,

what he started seeing was like, he started seeing, not just fatty liver disease, but in general, kids are getting hypertension high cholesterol, not just associated with diabetes. Like, they're getting them early and earlier and he was seeing that kids are getting diseases of their grandparents before their parents. So when you ask Jeff, our kids aging faster and you can't even finish the sentence. Absolutely. Absolutely. Our children are aging faster than they should. And

again, getting back to this, remove, we've been moving kids and young adults away from, you know, whole fat malls and the plant-based malls have no C-15 in them. Of course not. So then what, where else would you even get C-15 if you're not getting it from high fat dairy products? Are you

“even able, by the way, before you even tell me that, can you get C-15 from 2% milk or non-fat milk?”

You can, that's a good question. So whole fat milk has, if we, so in the 1950s, we drink two cups of whole fat milk per day Americans. And so we were able to get the amount between 100 to

would be equal to whole fat milk. But if you drink non-fat milk, no C-15. And if you drink

low fat milk, it has half the amount of C-15 is whole fat. And if you're drinking whole fat milk from cows that are fed corn versus grass, that corn fed, the, the milk from corn fed cows has 50% less C-15 in it than grass fed. So we've got changes in the, we have so many things that have been driving away decreasing our exposure, healthy exposure to C-15. Oh my gosh. So then, what other, okay, now you can add to the other pressure. So where else can someone, how, what,

where else does someone find C-15 if it's not in high fat dairy products? So that by far, this is how we're able to successfully get up and over, was now being increasingly defined, not just by us, as C-15 deficiency. So we've got to get up and over that 0.2% on the population, global population is sitting, the average is 0.2%. So we're on average sitting right at that precipice of cellular fragility. Wow. So, so the whole curve has shifted. If you look at C-15 in mom's breast milk

from the 1970s and 1980s compared to now, it's about half of what it used to be. So even from birth, our babies are getting about half the amount of C-15 because they're not drinking milk. That's right. So because mom needs to get enough C-15, you have it in her milk to get it to her baby.

“It's a ripple effect of what's happening. That's why you were saying earlier why the younger generation”

is getting all of these like very aging diseases more than their own parents because at least the parents were drinking milk because it wasn't, it wasn't villainize as much. That is extremely interesting. Yeah, and it's, you know, again, that, you know, it's promising about it is, we have, there's so much data in front of us now as you just can't ignore it. So we really need, we need, you know, ability to be able to take a good look at this data, remove our blinders on

saturated fats and say, yes, there are differences and how do we get saturated the good healthy saturated fats back into our diets through good agricultural practices, healthy, you know, wholesome milk in our younger years and then now with the navy funded us to do was to say, okay, how do you, in your older years, you know, as we, as we get out of our teens and move up, how do we get pure C-15 delivered, absent of the pro-inflammatory saturated fats in, in dairy

fat, is it something that we could help to actually optimize our C-15 levels to the only into those the longevity space. So we can do both. Let's fix the question. So you were able to figure out a way to kind of extract the C-15 molecule from the dairy, the saturated fat. Let's just say the dairy fat. If you're someone who is lactose intolerant or can't eat the butter's in the whole milk or

“whatever, you can still optimize by taking like fatty-15. That's right. So in that's why it was”

developed. So we used a plant-based C-14 and we had a carbon onto it so that, and our approach was that we were developing this to see if we could have it as a treatment for fatty liver disease. We were thinking about this from a pharmaceutical perspective. And so the robustness of all the data that we would need to do for pairing this to help treat a disease. That was the first five years of work.

It was at that level of research. Once we realized it was meeting the criteria of essential,

which means that we need certain levels of it in our body in order to just stay healthy. Like, to not get sick, then our bodies don't make enough of it. Therefore, we have to get it from our diet. That then tipped it from, this isn't a pharmaceutical, this isn't a drug, this is an essential nutrient-affoundational nutrient we all need. This can't be via prescription,

“this needs to be, you know, more accessible to the world. Can I ask you a stupid question?”

I've been wouldn't do it anyway, but like, how? So, I know, and so on. So, you'll be okay with it, but I mean, maybe you've been asked this before, but like, how is C-15 different than like omega-3? Right, okay, that's a super good question. So, because if you

know, we talk about this discovery of C-15 and, you know, as an emerging essential fatty acid,

that it would be like saying, you know, we discovered vitamin E, and how does that compare to vitamin A? So, the omega-3 is omega-6, that there are essential fatty acids, and so they are also essential, and they play role in helping to keep our cell memory because they have these double bonds in them, which are like hinges in the structure. Their job is to help make our cell membranes more flexible.

15 chain, 15 carbon chain. It's job is to keep our cell membranes stable. So, it's like a yen and yen, where they work together, where we need both flexibility and stability in ourselves. So, when we focus leaned in hard on just to make it three, and then we actively took, see, 15 out of our diet, then that created an imbalance in what we need for ourselves.

“Because I think that some people who would be listening would be, like, that's why I asked the”

question, because it sounds like when you see fatty, 15, or, you know, what you're at essential,

fatty, molecule acid, whatever it is, you ought to matter, okay, think while I take omega-3s, because that's again, considering that's been drilled into our brains, right? Like, there are certain things that over time, we've been conditioned to think that are essential, right? Like vitamin D's, it's essential now, omega-3's essential now. Basically, those are the two here all. Now you're hearing a lot about creatine as, like,

as a supplement to take, but like what I've noticed is like, if there's so many things that just because it's not popular or people by the masses don't know about it, it doesn't mean it's not essential. And in fact, a lot of times, they're just as if not more important. And so what I,

what I even notice is people take their blood tests and then they think, okay, I'm going to take

“these supplements, and then they can do that for the next 20 years of their life, right?”

And they're probably not even deficient in those things anymore, and then they get all of these different diseases or ailments or problems, and they don't even know why, because they don't even know what to look for, right? So like, is there even a blood test? Is there somewhere that people can be like, hey, can you check my C-15 levels? Can people even do that? They can. And this is such an important question, and it really gets to the heart, right? Of

being able to use science that, you know, high quality science to get us to where we are today, and then be able to transition that into the clinical practitioner space, right? So it's like, even for ourselves to take, you take care of our own health and have ownership of it, that you can measure C-15 levels. And so we worked with Janova, and they, so they have, it's been included as part of their neutral valve panel for a long time. So I was really,

yeah, so I was talking with, you know, Dr. Mark Hyman, and we were on a podcast, and I was like, yeah, it's on this, it's on the neutral, it's like, what? And so, so he went back and he went back and he was like, oh, why are we not testing for this? We need to get them to do that. So he went back and he looked at, and he's like, I cannot believe. He's like, so it's been collected all along. He's like, I went back and like, I cannot believe how many of my patients

are low in C-15. Nobody has just known, nobody's known to look at it. Right. So there's actually been a good number of people who have Ari have test results on C-15. So if you had a fatty acid panel, then they don't all include it. We're trying to get, make sure that they are all including C-15 in that panel. But for folks like Janova who do include it, they're able to measure it. We worked with them to, they developed an at-home blood spot test. So you can measure C-15 that way too,

because we think that's important. This is not just saying, hey, totally cold off and discovery. Yeah, it's like, it's like, what's been so important about this is, you know, we brought on Dr. Nick Schorke from, he has been head of NIH's longevity consortium for over 20 years, Jen. So he's seen every longevity nutrient or even molecule under the sun. And Nick said, I am watching the preponderance of data that are supporting C-15, like from all different ankle cell-based studies,

animal studies, human studies. He's like, I have never seen a molecule have more support for its

core role in supporting longevity than C-15. And he specifically said, this is going to be a geroprotector. I was like, great. What's a geroprotector? Yeah, I was like, well, that was a rarest. He's really excited. But it was, and so he said, your geroprotector is a molecule that can effectively slow our biological aging rate to stem the onset of the diseases that kill us. So it's not about you just tap into a mechanism of longevity. If you're a geroprotector,

you're going to have meaningful benefits within months. And he's like, this is the Holy Grail we're looking for. Not for, because to do a clinical trial that you don't feel or see any benefits, and you just wait and see if you live longer is, yeah, it's just not going to be. No, exactly. So, but with C-15, he's like, this, this is checking off all the boxes of a geroprotector. And

“that's why when we brought this supplement, you know, 50/50 to the world, again funded by the”

Office and Naval Research to do so. Then it's because it's intended to improve our, the strength of ourselves that then translates into improved health and wellness within weeks to month. So we have

16 weeks. Plus thank you. It's stop sneezing. It's amazing. No, it does not stop sneezing, just

“right. Yeah, yeah, it's a fantasy for everything actually. It's not fantasy. But and and what's”

most importantly is with regards to health, you know, within people go to their doctor and we go to their doctor, they're like, what are you doing? And they say I started 50/15 and it's those benefits. So we encourage people start on fatty 15, get your get your C 15 levels. If you can't see where you're at on the spectrum, are you deficient? So your goal is to not be deficient. Right. Or are you in the middle. And now you want to optimize to like, serdynians have C 15 levels that are

two to three times higher than S. So it's like, how do we optimize to get to serdynian levels?

So because they're drinking a lot of full fat, I bet you Europeans in general, because they don't

have the same nonsense over there with the food with like, when you go and get a latte or you get, you know, you get cough, people are drinking whole milk there. They're not asking for the oat milk, and this macadamian and cashew milk. I mean, this is very, very much like a North American thing. That's right. But wait. So because I want to ask you where else you can get C 15. So I know that we couldn't jump that. Yeah. But while we're still talking about this, I wanted you to tell us

besides the things that we talked about, which is diabetes and, you know, what you're saying

“about, you should diabetes, of course, fatty liver disease, blood insulin levels, what are some”

other symptoms or things people can look for that may be that C 15 can help with, and that it helps with like that you know that you've researched. Right. So for that, from the, it's the best ways we had the, the blood tests. Right. So the blood tests that there's specific things you can look forward to actually see signs of cellular fragility and the way that we look to see our ourselves

fragile because our C 15 levels are low. So first test your C 15 levels if you can, but not

everybody has access to that. Right. So then second is when you get, you can get a, it's standard to get a red blood cell panel. And that's where they usually look for signs of anemia. Right. And so if you have a low hemoglobin and increased amount of what's called red blood cell distribution with their RDW, that means that your red blood cell size is, there's a lot of variability, which means the cells aren't stable. So the cells are dying too fast. You're needing to make new ones.

And that creates a lot of different sized red blood cells. RDW has come out as one of the strongest predictors of biological age. So it's feeds into what's called age Excel, you know, which is now

“gold standard measurement of our biological age. And so RDW is important, not just for cellular”

stability, that's stability, then market becomes a market for how old we are biologically. So we need those tables. So RDW, when we get to then this pharoptosis. So if you're progressing to where C 15 deficiencies leading to this new form of cell death, then your blood results will show up high fared in level. So hyperpharotinemia would be something. We see a lot of physicians light up when they hear they're like, oh my god, I have a solution for what why I'm seeing this and so many

patients, like I'm seeing this combination. I haven't known what it was about. But what is it like if I don't know all the science stuff. And for me, just like a layman person, like talked to me like I'm like 11 or 10. Okay. Are you ready? Yeah. Okay. So we're going to do two levels. We're going to do adult going to their doctors and they get their blood results back. Okay. So you're going to see high cholesterol, chronic inflammation, signs of insulin resistance. You might see higher glucose levels.

Yeah. Elevated liver enzyme levels. So called ALT and AST. So these are standard things that your doctor is going to send you back and get like, uh, and this can help. So you 50 can help with all of it. It is, if it is part of this whole issue with regard to accelerated aging, yeah, the aging part. So also because I heard for skin, I have eczema. Yeah. I know that that's a big thing as well. Can you talk about how it can even help with skin issues? Because to me, like I have, I get eczema

dermatitis, like a topic dermatitis. It's a real problem. And I can't control it. Like I do all the things right. And yet it still flares up and it doesn't know people are like, sorry. You're going to put some more cortisol on your arm. Right. You know? Yeah. I have an actually right now over here. I'm like you're pulling it off pretty well. So thank you. Thank you very much. I'm covering it like this, you know. Yeah. So I have also, I also have a topic dermatitis. And it's from, if I eat any

raw fruits, vegetables or nuts. But it triggers it in allergic response in my skin. So I don't know. Wow. Which is like anything healthy. So make a joke in the book that I have a diet of like a six-year-old

So yeah, I thought you have to find a way. What helps you for your C-50? Yeah. Very old. There you go.

“All right. So about a six-year-old birthday party. Yeah. Your next diet bugs. That's right.”

Yeah. So with regard to, when we ran C-50s, we look at how to C-15 work. It's called these mechanisms of action. And so we talked about you how to strengthen cells. It also inhibits M-Tor activates the M-P-K. So those other things we talked about, wrap up my cement forming up. So it's called a, it's called a pleatropic molecule, which is just a fancy way of just seeing it gets around. So, you know, it does a lot. It does a lot. It does a lot of molecules. Yeah. Yeah.

Yeah. Yeah. Exactly. It's one molecule. That will be the title. The podcast. C-15. That's true. With a thumbnail, actually. That's right. That down. Yes. So it's, so it, it does a lot of things,

“which an essential nutrient should. One of those things is it helps to inhibit what's called”

jackstats. So it's a jackstat inhibitor. This is the mechanism that's being used for a lot of drugs that are being used for things like eczema and for atopic dermatitis and for psoriasis. So when we were doing those early screening of C-15 back when we were, you know,

assessing it for a drug, one of the first things you do is say, okay, what are its different

mechanisms of action? And that is where it revealed that it had this like anti-alogy anti-autoimmune disease activities. So to be clear, the supplement is not to treat or cure any right diseases, but really interesting that, you know, as we saw this dose response effect, that the higher the C-15 went, which we can achieve in our bodies, the stronger activity it had against these prone flammatory cytokines that cause what you and I feel with regard to experience with

eczema. So it calms things down. It's for me, it's helped to calm. It doesn't get rid of it. I can't go eat strawberries and have a great day, but it can improve it slightly or calms it down. If there's a lemon juice snuck into my cocktail, then I don't have a bad day. That's a wow. So, you know, it takes the edge off of it. And again, it kind of feeds into the question that keeps coming up is, we see a lot of these conditions have been increasing in the

last couple of generations. And so the question we're seeing is, again, if a lot of us are sitting on C-15 deficiencies, is that actually increasing our susceptibility to various conditions that C-15 was meant to be in our tissues to just help keep things balanced, to like keep our immune system from going out of whack or, you know, glucose metabolism and everything else. I want to take a quick break to talk about something I genuinely think as a game changer for healthy

“aging, and that's Faddy-15. Here's what you need to know. Scientists discovered the first new”

essential fatty acid in over 90 years. It's called C-15 and it's a big deal. When your cells don't

have enough C-15, they become fragile and they age faster. And when your cells age, your body age is full stop. As many as one in three people worldwide may have low C-15 levels. A condition called cellular fragility syndrome, and most of them have no idea. Faddy-15 co-founder, Dr. Stephanie Van Watson, discovered C-15 while working with the U.S. Navy studying aging dolphins. Based on over 100 studies, we now know C-15 is a key nutrient for slowing biological

aging at the cellular level. And Faddy-15 has three times more cellular benefits than omega-3

or fish oil. By the way, 72 percent of fatty-15 customers report seeing or feeling benefits

within 16 weeks. Things like deeper sleep, improved energy, and healthier hair, skin, and nails. It's vegan-friendly, free of allergens and preservatives, and clinically proven to raise C-15 levels. So if you care about your age, and I know you do, this is worth your attention. Go to www.faddy15.com/habits, and use code habits at checkout to get an additional 15 percent off their 90-day starter kit. That's faddy15.com/habits, code habits. And you can find the

link in the show notes. Now let's get back to the episode. I've got two questions. So comparatively, to let's compare it to a metaforement, which one is more

are an expert in C-15. I understand, but you've seen tons of data, tons of paperers and research. Where is the research strongest? I, on C-15, or generally for blunting blood sugar, to being the longevity molecule that is the king of all longevity molecules? Where would you say C-15 lies? So if I spoke for neck, because I'm slightly biased. So Nick, that's where we get a lot of our confidence is really coming from experts in the field. And so Nick spoke to

community of his peers in Copenhagen two years ago, and it was the largest anti-aging conference in Copenhagen, and he presented C-15, and he showed the data of why C-15 has the most support as a longevity enhancing nutrient, aka a geroproductor, than any other molecule that he has seen Barnett. And he's stood there, you know, unacologetic, and started out the talk we have it on it's on YouTube, but you know, you can go hide a link to it. Yeah, what's the called?

Um, it's, I don't know, it's just titled, but it's a known framework. It's called Nick with the, uh, title of the talk. A-R-D-D anti-aging research for the development of drugs. Oh gosh, it's a wonderful idea. A-R-D-D. A-R-D-D. But we'll provide you the link to that to the idea. I'm curious to see it myself. The other one I forgot to ask you about is N-A-D. Oh yeah, that's a good idea. I mean, like, N-A-D is like the hottest thing. It's, it's been

hot for a couple of years now, two, three years, but it's climbing in its popularity. Everyone's injecting themselves with N-A-D. Like, by the way, and I don't know if this is because my algorithm,

but now it's like every second reel is like, yeah, you know, I'm taking my injection of N-A-D,

and like, no one can live without it anymore. Because I know that you're, I, you're N-D to

“plead to after a certain age, and then every year it keeps on depleting, and that's what I've been”

taught to told many times that that's kind of the, the anti-aging panacea. What is your take on that compared to C-15? So N-A-D sets, so if you're like at this longevity, regulating pathway, right? And so it's a super complicated fancy-looking pathway. You put N-P-K activation is in the middle of it. M-Tor inhibition is like up here. N-A-D is right after N-P-K. So it's part, it's part of the longevity regulating pathway, N-P-K can help up regulate N-A-D. So N-A-D, so it can help extend longevity. So again,

all of the players in the longevity space are there for a good reason, and it's because they're tapping into these important pathways. So N-A-D, as far as, you know, being able to under it, so it makes sense of why it's there. And so, you know, in the clinical trials continue with it,

and I do think it's encouraging. It's less about it being essential because our bodies,

it's something that our bodies make, but then to your point, as we get older, the levels decline, and so to be able again hack into that, and be able to get our levels back up, is a good thing. Right. There's precursors like N-R and N-M-N, and all these other things. Right. But I've also been, I think that N-R is much, it's my opinion, but N-R is a better precursor than N-M-N for the size of the molecule, and all these other things. From what I've all the research I've seen,

but I wanted to ask you because you've, like, deep-dived on, of course, 15,

“and that's what you do for a living, and you've pricing a ton of data on a lot of things,”

and there's a lot of noise out there. So, like, if you had to create your, you know, the your top longevity cocktail, and it could be everything and everything and everything that, you know, exercise, of course, is a piece of it. Nutrition, what would you put in that cocktail to live, to live a healthy, long life, not just long, but healthy, and good? Right. And it's a

good question because when we first came out with the book, the longevity nutrient, a lot of people

Jen, especially women, they're like, "I don't want my javity. I want help." So, but if I can live a long time, and we help that whole week, it's that whole, it's just kind of sometimes a very different line for it. If you can live to a hundred, but be very, you know, in capacity, like, you know, completely like, that's not exactly a quality. I think how to live long and help with a

“quality life is the goal, right? Yeah. So, I'm going to sound real boring. Oh, this one is so bright,”

but I am a true, true believer. And, you know, absolutely like you said exercise in that includes, I mean, you know, not just aerobic, but as for resistance training, we're all understanding that now that to be able to be social. Socialization is huge. Yes. Keeping our hearing, we're now

that that decreased stimulation to the brain increases the risk of the earlier onset of

dementia. And so, that's not a, you know, a lot of repeated studies are showing this in, you know, we all end up losing a lot of us end up losing our hearing here or time. And it ends up that we really need that stimulation within our brain. It also, as we start losing our hearing, we become less social. And then you're as you're not interacting and being, you know, social with people, we know that decreases longevity and leads to depression, which again, short and slung

cavity. So socialization, hearing, exercise, sleep is huge. And if we don't, it's not just how much you sleep because there are some people who sleep too much. So if you sleep too much or you

“sleep too little, there's this Goldilocks zone in the middle, and not only do you need to sleep”

enough, you have that quality of sleep is good. Right? By the way, and everything, like, your social circle has to be quality, your food has to be quality, your sleep has to be quality. I think it's a kind of a give it, right? Yeah, just because it's there, just because it's available or you're doing it doesn't mean it's the best quality. You know, if that you're going to get the best results

basically. Exactly. And then, of course, C15 is in my stack. Like a hundred percent, I never took

supplements before this. Really? Yeah, I just, I'm not a good pell taker. And so I just didn't do supplements. And that's why you started the gummy. Well, I need to plan. So these, these capsules are super, super tiny, tiny. I think it's a small thing. I was like, where are they? They're like little micro, like they're like little bees, like little bugs. Yeah, which makes it really

“easy. So like 99% of our customers make fatty report that fatty, if you use part of their”

daily routine at six weeks, like at two weeks and at six weeks. So it's an easy habit that sticks. And then they stays. We have over 95% retention rate on it because people are feeling the benefits.

If 70% of our customers report feeling deeper, slate, comma, mood, better energy within 16

weeks. And we like calling those delights and surprises. This is really about the long-term benefits that delights and surprises are coming. We've recently discovered C15 has, again, about being a sweaty molecule. But it also has premiscuous, we'll go with it. Yeah, I like that's a better word. Nice humanism over there. Yeah, so that it also inhibits two enzymes. One called M-A-O-B, and the other one called M-O-B, and the other called F-A-H. And by doing having those two activities,

it helps increase dopamine levels and endocannabinoid levels. So by helping to boost dopamine and the effects of cannabis, right, endocannabinoids, that is explaining why people are like, "Oh, I'm sleeping better. I'm feeling better." Like, really. So the retention rate is really high because we had the added benefit of the gift that Dolphins gave us, which was, yeah, I actually feel better for a lot of people within a few weeks. So people make a part of their routine. So for me,

it's been, it's a non-negotiable and not because we're, you know, I'm coceo of the company, is because it's genuinely helping me feel better. Right. I said, put a few, if it helps sleep a little bit, or a lot of bit, depending, do you recommend people taking it at night or the morning or? Right. So it's, you know, for me, I take, I take them, the capsules in the morning, and I still get the sleep benefits. So which is interesting. We do have customers that say they'll take

one in the morning, and then they'll take one at night, or if they shift it to the night, that for the ones that are really like need that sleep benefit, that it's beneficial. So now that we have the gummies coming out, I, you know, I'll take a capsule one more. Oh, some gummies in there. You can take, you could double, you could double stack it. Yeah. Okay, I like that. Okay, I'm not finished because I have a lot of other questions that we haven't, we kind of like went

through, but we kind of kind of jumped over. Where else can you get C15 if it's not in dairy?

“Right. So dairy is our primary source. That's how we get up and over, not being deficient.”

However, there are other things we can do. Okay. All right. In addition to supplementation to get everything, everything we can do right. Yeah. Yeah. Improved. So we know that if we exercise, it increases our C15 levels. We think that's because it's when you exercise C15 is stored in your tissues. So as your exercise, it's helping to release C15 from your tissues and get it in circulation. Another added benefit for exercising. I mean, every day, there's like another thing that you get

from exercise. You'll see 15 boost in the blood. I love that. So that's good fiber. So there's a great study that was done showing that it was a great series of studies. And what they showed was that how is fiber helping liver health? And so they did a whole series of studies in mice. And they showed

Yeah. Okay. So where when you eat fiber inside a fiber is a molecule called angeline. So not insulin, but angeline. Angeline is then used by specific microbes in our gut to make C15. So our microbes, I can produce some levels of C15, which explain that even people who eat no dairy,

nobody has zero C15 levels. It's just about it's essential because you need a specific amount.

Yeah, to get enough from our diet. But eating fiber. So I have high quality cheeses, pecorino is what comes from Sardinia. So high C15 cheeses, pecorino is so sorry. Not to interrupt you, but I'm going to. Yeah. So this pecorino has the highest C15 content. Some of the highest I've seen. Really? Yeah. What other cheeses? A lot of pecorino. Right. Right. Right. Yeah. So like that. Hard cheeses. And those are specifically because they're from Sardinia. These are grass

“fed sheep. I believe the sheep's cheese. So grass is for sure. You said that earlier. Yep.”

And now the specific places where the highest C15 that you've seen is pecorino, cheese.

Let's see Swiss cheese. And then it just kind of becomes a mix of cheeses. They're kind of equal.

I would, the more concentrated and hard the cheeses, probably the fibers in other place. So fibers. So then our bodies can make fiber exercise, eating the high quality cheeses. And then there was actually a study that just came out two months ago that showed that people who ate more cheese, specifically of all the dairy products, had a lower risk of developing dementia. This is the largest study of it's kind of had over 27,000 people and it followed them over 17 years.

And again, it's association, but the timing, right. It's like, do they start by having, and they start by having a certain dietary change and do they see that outcome over time.

“So it's interesting. Wow. You know, there are these parallels. So again, it's been two”

absolute. It's been two. Don't eat their whole fat dairy products. There's just differentiation in some of that differentiation, maybe from which foods have the most C15 in them. You know, I also, as you're talking about this, do you have a patent? Because like, can't people just replicate this? Like, how do people know, like, you would think with 100 now peer-reviewed studies, that's a lot that, you know, there's a resilient supplement

companies out there, right? Like, what's to say that, like, tomorrow, I can just go and be like, you know what, I know all about this and I'm going to create a pill and put some C15 in it and call it a day. Can't people do that all day? So they can't because it's the Navy. So because the Navy, the Navy, the Navy, the Navy, the Navy, the Navy, the Navy, the Navy, this discovery. So I made the discovery while I was at the Navy. So the, the patent is around the use of C15 to be able

to have these benefits. And, you know, there's, there was a time where I was like, gosh, kind of feel about this. Like, this is for doing proof-global health. Yeah. It's a patent. And then I feel really good about, and this is why is that when we have seen, you know, competitors come to the market. Yeah. And they put their C15 supplement out, Jen, we go and test it. There's either no C15 in it at all. Yeah. Or the C15 in it, the quality is terrible. Terrible.

And so the patents are actually protecting the highest quality and not, it's preventing or allowing us to be able at least try to keep this massive number of, you know, it's like, whack them all out there. Well, I talked with a lot on my social media. And I talked a lot about this in just all my shows, because I feel like because it is the Wild Wild West, you have no clue what you're getting online. You've no clue. And I've even tested multiple things. And was really shocked at the limited amount of

whatever that ingredient is that they're selling as like the ingredient. Yeah. And because you just, because people don't know. And that people think if it has, if it has a lot of reviews on Amazon, oh, that must be a good thing. You can buy reviews. You can do all sorts of wacky things online. So like really knowing where you're getting a sort, this is for everybody listening, know and research where you're getting everything from. Because it is dangerous out there.

You can put just, or at the very best, you're just having like a bunch of dust or like flower or whatever. Right. And that's been, yeah, I'm so go ahead. No, as you see, it's very, that's why it's very important.

“But that's why I wanted to know, like, because you have to have some kind of restrictions around”

what people can do with what. So they don't just go up there and make and create a bunch of

baloney, basically. Yeah. It was, I mean, maybe super dived. Yeah. I had, I never would have thought

And you believe it. And it has none in it. But that's because people don't expect to

“hide even like, right. And now it's like, so it is. It's incredibly disappointing. It needs me”

glad there are patents to pressure like to say like this one, you know, and therefore, you know, it, and then we're, you know, participating in programs like subco, right? Subco is, you know, they do measure on 26 different levels of quality. And so we have a subco score. That's really good. So it was 9, 6, 3. Well, no, and a couple weeks ago at 9.88,

of out of 10, which is in the excellent amazing. And so the ability to have these platforms,

so that exactly like you're saying that we need consumers, we need to build a, like, separate the wheat from the chaff. And because it's, like you're said, it's not only what's not in there. It's what is what is the thing, just in case. So anyway, I'm glad they're out there. We're obviously thrilled to be participating in the programs because it gives us a chance to say, not just this great science and the reason why it works is because of the quality of the ingredient

that's in it. And all the science that's been dedicated, but we're also going to uphold. We do third party validation for, right, for its quality. We had an independent group, assess that it was generally recognized as safe for one year and up. And we even had an independent team come in of scientists come in, evaluate our claims and determine and reconfirmed that they're based on competent and reliable scientific evidence called cars. So it's just like, for us,

“it's just so important to be able to say, yeah, how do we separate ourselves from all this noise?”

There are ways to do it. So yeah, you know, that we're owning that. That's really good. Okay, so let's get to the dolphins again, because I know I kind of skipped through that a little bit and more question to the dolphins. If you don't mind, do you mind if I keep you here another few minutes? Yeah, not at all. So you're going to talk about, you don't like dolphins. No, I love dolphins. Who doesn't like a dolphin? I mean, there's such a beautiful animal.

But what I was going to say is, where did they find, when you were looking at all these dolphins, and you said, some had higher C-15 than others, they don't eat cheese. And they're not eating those fatty fishes like the Yukon fish that you were mentioned earlier, where were they getting the ones that had the higher C-15, where they, they have a higher C-15 because of their genetics or because of their muscles,

it was releasing faster, like you were saying, yeah, they eat more fiber, like how were they higher and C-15 than the other dolphins? Yeah, so what happened is when the Yukon went away, then the Navy went to five different types of fish. So within that, so then they were getting, you know, herring and a capelin and squid and macrol. So what we found was, and then based upon preferences of the dolphins, they would get more of one versus another. And so we kind of had,

even though we think fish are fish, they're not. And that's some of the fish called capelin. There was tiny little fish which were great, and they loved them, but they ended up being like the chips of the sea, right? It was like they had like no C-15 though, really. So yes, they, the way it differentiated between some dolphins that were getting more of versus not, which is a great question, was based upon how, which dolphins were getting more of the macrol

and herring that had higher levels of C-15 versus the percentage of their diet that was getting, like, oh, a lot of cablin and squid. For example, had no C-15 in it. So macrol and herring have a higher C-15 than other fish. Yeah, and mollet, you know, the really popular fish.

Yeah, I've never heard of mollet in my life. It's been hair cut. It's been hair cut.

It's been hair cut. It's been hair cut. It's been hair cut. Yeah. Yeah. That's more of it. Yeah, some mollet fish. Okay, I'm not going to apply it on needing that. So just tune on salmon. Do they have C-15? They have C-15 in them salmon has a d-level mega, right? Yeah, exactly C-15 as a decent amount in salmon. When we tested the fish, it's in the highest levels in the skin and

“the heads of the fish. So I think that's why it ends up for us not being, there have been studies”

saying, can C-15, you know, we know C-15's a biomarker of dairy fat could it be a biomarker of fish because it is in our fish and it's, it's not strong at all. And I think it depends on what type of fish are eating. And, you know, if you're a Scandinavian or Norwegian, you might be eating the skin and heads of fatty fish. Oh, yeah. So if it's anchovies and sardines and then Asian cultures, you know, used it traditionally like the person of honor at the table we get the head of the fish.

Yeah. Little did they know it was because they were getting C-15. Yeah, exactly C-15. Exactly. Okay, I have another question about C-15. So if it's so good for anti-aging and kind of inhibiting the acceleration of your age, why are beauty products not using it yet? Are you going to

be making beauty products? Because they always jump on every bandwagon. They bring everything in,

like is it top, can it be used topically? Like if I opened up a supplement of fatty 15 and just put

help. I mean, I don't know. I made a nice mask. I think that's about it, you know, but I was wondering

are there other ways that people, you know, can other industry because I would think it is such a, like, is that like a product line that you would make? Yeah, it could be kids and I know you come with kids stuff, right? Because I couldn't believe that like you were saying how many kids are not

“getting enough of this very essential mineral, what do you call it, molecule? Because their parents”

aren't drinking or eating it now. Right, crazy. And I don't know if you remember, but like when our send-in when Ben turned to, we got the paper sheet of paper that said, okay, it's time to move your kid off of whole fat melts on it. Yeah, and it was like, it's not like he was overweight or he had diabetes. Like, so, you know, we kind of, you know, was this before all of the just happening, but it's like that has just, so why, why are we taking, oh, yeah, that was absolutely fantastic.

You know, and by the way, I don't know if you, like, I do notice that whenever you take away you go from like an alternative, like, you know, the whole thing was like, mirror snack wells, like, you know, like, like, bring back everything, everything has its like trend, like low fat, hot, you know, this thing, that thing, like whenever you take out one thing and you substitute it,

it's never good for you. That's right. Right. So why do people even do that? I don't even know.

A cold milk, the people I know who drink cold milk, they are very lean, they're thin, they're they are healthy. There's just crazy psychology around, I'm going to get fat if I have these whole, whole milks and whole foods. It's a really, it's a, I know you're not as psychologists, I'll, I'll, I'll take that up with somebody else, but I just find it interesting how sometimes, like, leaving something that's natural state and using for what it's utilized for it's the

“best way to do it. Yeah, I, you know, completely agree. And at some point, it'll kick in, right?”

We just need to keep everything balanced. It's just, we just need to, like you said, don't do these extremes or you're taking one thing out in place, it's just balance. I know. All it is, it's just keeping, and exercise, and exercise, doing, and exercise. I want to ask you one, I didn't, we didn't write touch about, but we talked a little bit, but is there anything in this book that is, like, because we hear so much about longevity, right? Is there any, any other tidbit that you can share

that maybe we haven't heard 400,000 times? I like to, because I, I'm always looking for, like,

that little, not yet, that might be a little unique, right? You know. So, by the way, this is why I wanted to talk to you about C-15, because I have never heard about it. I wanted to kind of tell that to be able, because we hear about the same things at nauseam, and we, and whenever there's something that has been a little bit less covered, I want to bring it to, you know, the ethos, or like, my audience, at least. So, what else can you share? That's a little bit more unique than

what we've all heard. Right. So, this goes over, you know, obviously the book, the focus of it, is on, it's to increase awareness of exactly that. You know, of C-15, it goes through the dolphin discovery story, it goes through the mechanisms, like, how do we, so it does, you know, something that's new is going through what a general protector is. So, like, how do we differentiate, right, between, sure, it taps into this, here are the, you know, 12 homerics of aging, here are the

mechanisms of action. So, those, and they're still in there, but it's reframing to say then, what are the things that we should really be paying attention to? And it's the ones that made the criteria of a general protector. And so, this is a list that, you know, every consumer could actually work their way through and say, gosh, is there evidence that that's more like it all? Does, and it doesn't, it doesn't have to be the deep science part, it's just like, wow, is it actually going to

help change my cholesterol glucose? You know, like, things that I care about, right, you have so many people with comorbidities that they have this, you know, multiple diseases at the same time, because of aging. So, it's like something that then, again, a geroprotector, right, the whole name is not very sexy name, but it's okay, but it's, it's factual, so it protects us from aging. So, by doing that, it can help address, right, these multiple components that lead to having these

multiple chronic conditions. So, it's a new way to challenge interventions, and to say, like, how we should be thinking about it isn't just, you know, sexy longevity science, like, ooh, here's something cool, because it taps into this pathway and it helps warm, which is still exciting, but is it going to make me feel better, you know, in kind of, you made a really good point of,

“I've been taking this supplement for 10 years, why? Yeah, well, I think that's majority of people,”

right? Like, why? Unless you actually asked the question or hear some, I said, yeah, why? That's why. Like, what I, I just did, um, and I'm going to do my blood panel again, but I just did one and a few, like, a couple months ago, three months ago, and all the things that I've been

because I thought, okay, I was deficient in vitamin D 10 years ago, so I kept on taking it,

and guess what? Now, I'm over vitamin D, like, I have too much the levels too high. And so,

“that's what I'm saying, like, just because you hear it in the ethos, does not make it true for you,”

particular, right? And so, that's why I'm urging urge everyone to, like, a get their levels checked regularly and see where you are, and then it would base where you want, like, kind of reverse engineer, like, do you feel better? Is that improved because you're taking this, not that? So, if you guys have not, you know, checked your C 15, I would urge you to do it, because that could be the reason

why you're having an ailment that you never even, like, connected the dots to. Yeah, that's right.

And so, that's the whole purpose of behind this is it's, it's data driven, you know, nutrition, and you can personalize it. So, and you can see, do my levels go up? Do I, did I move from deficient to sufficient, did I move from sufficient to optimal? And most importantly, am I seeing or feeling benefits? Right. So, give yourself six months, and then if it's like, it's not really

“do anything for me on my blood work or how I'm feeling, if you don't need to take C 15, right?”

Right, if I could, it's just like, we've really challenged people to, you know, get used the data to drive to say, is this the right thing for you? And what we're finding is in most cases, you know, again, the 95% retention rate, people are like, this has changed my life. Yeah, I've had, like, I've had a lot of my friends say really positive things. It's helped them a lot, and like, I think at your, like, to your point, I think once you start taking it, I think that

what people have noticed at the real improvement. Yeah. Yeah. And then when they go off of it, so sometimes they're like, that's another great thing. Like, they're like, I'm not sure if it's helping me, and then they're like, and then I went off of it, and I was like, oh, yeah, yeah, that was helping me. I was helping me sell our back on it. So, and that's another test. Right. And so we really encourage people to like tailor for what's good for you. Yeah, you know, I love the fact that this is like,

it's new to me anyway. Maybe it's not new to everybody, but it's new to me. And so thank you for being on my show. And thank you for telling me and talking to me about this. Is there anything else I haven't asked you that I'm missing in dolphins or, like, you or besides your crazy resume of bad assory? Is there anything else that we can talk about and tell people? I love, and I don't know how many bad assiners are nerds that there are out there. There's a lot of bad assiners. I know that

that's true. Um, about to check my nose. Yeah. I, you know, for, for for me, Jen, Jen has been the absolute pleasure chatting with you. My, you know, this is, this is a movement. The dolphins gave us a true gift. Right. And again, this whole new perspective to be able to look at things, 10 years over a hundred peer-reviewed study since then is now lighting and something we can go along right now. We have an opportunity to help truly improve, truly improve global health.

And we aren't alone anymore. You're like, you know, it's now it's just, we have a giant community of scientists and doctors and, and, you know, moms and dads and kids. Like, it's just, it's, it's the movement is on, you know, part of it. So thank you. Oh, and it's just, I like it. I appreciate

the, I appreciate this because like I said, I always, I was telling you earlier, I always look

for ways to sneak health into my children because if I tell them it's healthy, number one, they'll do the opposite because they're kids and they all never believe anything I say. So I'll just tell them it's junk. And now that you're making a gummy for kids, it's a, it's a, it's a

“great little sneaky way to kind of get a very essential molecule into them, which then helps them.”

Because now I didn't even know the amount of kids that are actually deficient. So you just did me a favor. So thank you. Thank you. This has been a pleasure. No, pleasure is all my and guys. So check out Fatty 15. Check out Stephanie. She is, I'm telling you, just, just deep dive and you'll be super impressed like me. So anyway, thank you for being with me today. Thanks, Jennifer. Okay. Bye.